0000-0002-5766-679X
Open Letter: Scientists stand up to protect academic whistleblowers and post-publication peer review.
robustclearexciting
A Novel N-terminal Region to Chromodomain in CHD7 is Required for the Efficient Remodeling Activity
Overarching control of autophagy and DNA damage response by CHD6 revealed by modeling a rare human pathology
CHD7 regulates cardiovascular development through ATP-dependent and -independent activities
The DNA repair protein SHPRH is a nucleosome-stimulated ATPase and a nucleosome-E3 ubiquitin ligase
ortho-Fluoroazobenzene derivatives as DNA intercalators for photocontrol of DNA and nucleosome binding by visible light
EcR recruits dMi-2 and increases efficiency of dMi-2-mediated remodelling to constrain transcription of hormone-regulated genes
Assembly, remodelled.
SIRT6 recruits SNF2H to DNA break sites, preventing genomic instability through chromatin remodeling.
Chromatin remodeling by the CHD7 protein is impaired by mutations that cause human developmental disorders.
Methylation-sensitive single-molecule analysis of chromatin structure.
Analysis of individual remodeled nucleosomes reveals decreased histone-DNA contacts created by hSWI/SNF.
ATP-dependent chromatin remodeling complexes in Drosophila.
dMi-2 chromatin binding and remodeling activities are regulated by dCK2 phosphorylation.
The dosage-compensation complex in flies and humans.
The dMi-2 chromodomains are DNA binding modules important for ATP-dependent nucleosome mobilization.